Sentence examples for uptake component from inspiring English sources

This protein nanopore was based upon the native scaffold of the bacterial ferric hydroxamate uptake component A (FhuA) of Escherichia coli.

Recently, we redesigned ferric hydroxamate uptake component A (FhuA), a 22-β-stranded protein containing an N-terminal 160-residue cork domain (C).

A truncation derivative of the ferric hydroxamate uptake component A (FhuA), which featured the deletion of the 160-residue cork domain and five large extracellular loops, produced the conversion of a non-conductive, monomeric, 22-stranded β-barrel protein into a large-conductance protein pore.

Subsequent substrate uptake, especially palmitate uptake, has to be corrected for the non-protein-mediated uptake component.

The non-saturable uptake component can be estimated from the uptake in the presence of an excess of substrate.

This uptake component was abolished in LRRC8A−/− and LRRC8(B,C,D,E −/− cells which do not express functional VRACs and was inhibited by the VRAC inhibitor carbenoxolone.

In addition, we found CRE1-repression of nitrogenous substances uptake, components of chromatin remodeling and the transcriptional mediator complex, as well as developmental processes.

By contrast, the low virulence after the inactivation of siderophore-assisted iron uptake components, sidC, sidD, sidF, and sidG indicates that siderophore biosynthesis but not reductive iron assimilation mechanism is essential for A. fumigatus virulence [ 82– 82].

Therefore negative regulators are required to balance the induction of Fe uptake components to reduce the negative effect on plant growth that results from excessive Fe and/or as a result of the induction of the components due to Fe deficiency.

Moreover, about 50 additional genes were assigned to iron-siderophore and ferric iron uptake components, which have few orthologs in other corynebacteria, indicating that the acquisition of iron is a key factor for growth of C. variabile on the surface of smear-ripened cheeses.

Genes of the neutral bile acid biosynthetic pathway (Cyp39a1, Hsd3b7, Akr1d1, Cyp8b1, Slc27a5, Acox2, Scp2, and Baat) were repressed as were the bile acid secretion and re-uptake components Abcb1a and Abcg5/8, respectively.