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Quantitative in vitro studies have helped elucidate mechanisms underlying processes leading to functional biomechanical changes.
The important question is what are the underlying processes leading to this contrast.
This alternative is based on the suspicion of two (or more) underlying processes leading to an event.
As our research did not attempt to clarify the reasons for the denial of care, additional research should shed more light on the underlying processes leading to this phenomenon.
Much recent research has been aimed at understanding these early underlying processes leading to dysfunctional synaptic signaling, as this knowledge could identify putative sites of interventions, which could potentially slow progression and delay onset of disease.
Much recent research has been aimed at understanding the early underlying processes leading to dysfunction at the synapse, as this knowledge would likely inform interventions that could potentially slow progression and delay onset of disease.
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This improved outcome may be secondary to increased levels of monitoring leading to earlier detection of cardiac arrests in these patients, or may suggest greater reversibility of the underlying process leading to the arrest (for example, myocardial ischemia or altered autonomic tone with spinal cord injury).
Prompted by some of these observations we implement a conflicting dynamics model that generates this distribution and contributes to the identification of relevant underlying features of processes leading to it, as well as to a characterization of its parameters.
However, the contrasting anatomical characteristics of gymnosperm and angiosperm xylem may underlie different healing processes leading to the lower regenerating capability of the former.
Studies focusing on clinical events cannot distinguish between the triggering of acute events and chronic processes leading to the development and progression of atherosclerosis as the underlying pathology for cardiovascular events.
Since human hepatoma generally occurs in patients of chronic hepatitis with/without cirrhosis, to identify the genes implicated in the common biological processes leading to hepatocellular carcinogenesis, we selected three hepatoma patients with different underlying liver disease.
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