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The ultimate methyl donor for epigenetic-methylation reactions is the folate-methylation cycle and feeding pregnant dams diets deficient in methyl donors results in altered epigenetic regulation of specific genes in the offspring; e.g. axin fused [48] and the Agouti gene which is under imprinting control [49], [50].
Under imprinting, then, patrigenes should favor more investment in sisters than matrigenes favor [ 14].
The covariance between additive and dominance terms is strictly negative under inbreeding alone, and is on average negative when averaged over males and females under imprinting alone.
Under imprinting, the breeding values and dominance deviations are no longer uncorrelated, which means that the total genetic variance cannot be partitioned into the usual additive and dominance variance [ 19].
Therefore, for the sake of clarify, we will call 2 p q(a+(q− p) d)+2 p q i as the additive genetic variance, because this is the variance between breeding values under imprinting.
Recall that under imprinting alone σ A D f = a p 1 p 2 α f (k 2 − k 1 ) and σ A D m = a p 1 p 2 α m (k 1 − k 2 ) (Table 3).
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It is shown that CRC structures which are initially under-imprinted may be brought to a resonant condition by increasing the metal deposition thickness, which is desirable for improving the thermal and mechanical properties of the structures.
As the heated mold comes in contact with the resist, the resist will be heated up and soften into a molten stage, where it will fill in the mold cavities under sufficient imprinting pressure and time.
Loss of imprinting under the coadaptation theory has not been formally explored.
Thus the mammary gland is an obvious target for imprinting under the maternal infant co-adaptation hypothesis.
Keeping this major caveat always in mind, we can still attempt some predictions about the patterns of loss of imprinting under each theory.
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