Although the spatio-temporal origin of the outbreak (acute care vs. LTAC) remains undefined, these findings likely reflect longer exposure to the continuum of care and higher rates of co-existing conditions within the LTAC population.
Although the antigens in the IHA are undefined, our finding suggests that these polysaccharides may be two of the major antigens in this assay.
1 Although a genetic component in the aetiology of major depression is evident with a 40 50% heritability, 2 the predisposing genetic background has so far remained largely undefined, and recent findings from genome-wide association studies also point to a complex underlying architecture.
It is conceivable that Mendeliome could be preferentially applied in clinical settings with largely unspecific and undefined phenotypes and/or misleading biochemical findings (which we personally experienced in this particular case).
The present findings indicate that undefined cells, committed progenitor/transit-amplifying cells, and stem cells that take up nuclear label during allometric growth of the mammary gland and retain this label continue to cycle throughout pregnancy, and that ER-α and PR expression in these cells is affected by pregnancy.
Results of this case series indicate nonspecific findings or findings of undefined significance.
The high level of CR1 in aetiologically undefined pneumonia is consistent with this finding, since one can speculate on epidemiological basis [ 1, 17] that most of these patients probably had bacterial pneumonia.
However, investigations have been, for the most part, limited to small, genetically undefined patient series with rather variable radiological findings [ 4– 7].
These observations suggest that not only hyperglycemia per se but also insulin resistance and oxidative stress, or other as yet undefined factors, may be responsible for the findings in this cohort.
On opening the door, they find a dark, undefined space.
Taken together, the present finding has revealed a previously undefined role of CFTR and its mediated prostatic HCO3− secretion in the host defense against bacterial infection.
