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Although the precise mechanisms by which H. pylori induces carcinogenesis are still unclear, inflammation is the most commonly cited factor in the carcinogenic process.
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And it remains unclear whether inflammation might be implicated in all forms of epilepsy or which patients might benefit from anti-inflammatory treatment.
It is rather unclear how inflammation is triggered in the brain, which remains sterile under normal conditions.
It is unclear if inflammation interacts with stability-related factors.
It is unclear whether inflammation is the cause or the side effect of such conditions.
It is unclear whether inflammation contributed to tumorigenesis in these patients.
Underlying biological mechanisms are unclear, but inflammation and hemostasis are suggested to be involved (Brook et al. 2004; Peters et al. 1997; Ruckerl et al. 2006).
The exact pathophysiological mechanisms underlying the development of myocardial oedema in TCM remain unclear, but inflammation, increased LV wall stress and/or transient ischaemia (myocardial stunning) appear pivotal.
It is unclear whether inflammation is a cause or effect of hypertension, but as discussed in this review there is evidence from human and animal studies suggesting that inflammation can lead to the development of hypertension.
When the anatomy of the Calot's triangle was unclear or inflammation in the gallbladder neck was advanced, the bile duct stump was treated at the neck and the cholecystectomy was limited to a partial resection.
Despite a huge number of published papers on inflammatory processes during chronic neurodegeneration in the last 20 years, it remains unclear how inflammation contributes to progression of neurodegeneration (Wyss-Coray, 2006).
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Justyna Jupowicz-Kozak
CEO of Professional Science Editing for Scientists @ prosciediting.com