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In contrast, sedation, as measured by a decrease in motor activity, and the cardiac depressant and hypothermic effects, as determined by radiotelemetry, remained unchanged in the mutant mice.
The expression levels of both genes remained unchanged in the mutant within 36 hai (Figures 5J and 5K).
Expression of caffeoyl-CoA 3-O-methyltransferase (CCOMT) (spot 36), a critical in lignin synthesis and functions in structural barrier defense [6], was almost unchanged in the mutant, but was induced as early as 3 hai in Wangshuibai, peaking 12 hai.
Food intake remained unchanged in the mutant mice.
Contrary to the standard hypothesis, excitability of F-type motoneurons was unchanged in the mutant mice.
In contrast, the anti-apoptotic gene Bcl-2 like-10 (Bcl2l10) remains unchanged in the mutant oocyte, suggesting a self-protective mechanism adopted by the degenerating oocytes.
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The additional residues within 4 Å of the Trp203 side chain of wild-type MtDXR, Ser245 and His248, are unchanged in the mutants.
Thus, NAD+ binding is unchanged in the mutants, suggesting that the severe decrease in P5CDH activity of D779Y and D779W is not caused by alterations in the Rossmann fold domain.
The distance of the Asp228 carboxylate to N1 of the pyridine ring is unchanged in the mutants as are the noncovalent bond constraints on the phosphoryl group of the PLP tail and the coplanarity of the His148 imidazole with the PLP ring.
Transcripts of five of the 9 genes were unchanged in the ΔMovam7 mutant.
The induction of the gene encoding the chitin receptor LysM RLK1 was essentially unchanged in the atl9 mutant and wild-type plants.
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