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Due to the relatively low abundance of photosynthetic organisms in natural crusts, particularly in arid regions, we were unable to measure changes in O2 or CO2 exchange and hence could not determine whether the decline in fluorescence when the light intensity reached about 200 µmol photons m−2s−1 reflects a decreasing rate of photosynthesis in parallel with loss of fluorescence yield.
However, we were unable to measure changes of in vivo implant stiffness between pre- and postinduction.
However, this study was limited by the cross-sectional design and was unable to measure changes in health outcomes associated with the change in smoking status.
As we were unable to measure changes in MMP-2, MMP-3, MMP-9 or TIMPs following IL-17 exposure, however, we are unable to confirm that IL-17 acts through MMPs.
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We were unable to measure eventual changes in administration costs resulting from the reform.
Finally the study was unable to measure any changes in rates of deliberate self-harm or help-seeking as outcomes of the intervention.
For early life investment in fecundity, we were unable to measure significant evolutionary changes.
As our survey was only implemented at the end of the study, we were unable to measure directly the change in knowledge uptake or practice pattern.
In contrast, due to the HABAM and BI ceiling effect, these instruments were unable to measure clinically important change at the upper end of these scales for a large proportion of older medical patients included in this study.
We were unable to measure any short term change in distress due to the limitations of the trial protocol and by measuring distress at 12 months we missed any possible short term increase.
"It's hard to measure change.
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Justyna Jupowicz-Kozak
CEO of Professional Science Editing for Scientists @ prosciediting.com