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Incubation of the same cell lines with H. pylori BCF induced formation, enlargement and quantitative enhancement of structures positive for H. pylori toxins/virulence factors, NOD1, ubiquitinated proteins and proteasome, which closely reproduced cytochemical and ultrastructural patterns of corresponding in vivo structures, thus confirming a significant role of H. pylori in their development.
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They are characterized by lysosomal accumulation of autofluorescent lipopigment that display typical ultrastructural patterns (1).
The vascular ultrastructural pattern in terminal villi of placentae of this group showed no significant abnormalities and was normally distributed in the villous tree.
These corneal dystrophies and the sporadic disorder, iridocorneal endothelial syndrome (ICE), exhibit overlapping clinical and pathological features, including ultrastructural changes and abnormal patterns of cytokeratin expression consistent with epithelialization[4], [5], [6].
About 60 different, robust patterns of ultrastructural organization are recognized, but ~200 genera of uncertain affinities have yet to be examined [ 1, 4].
In the nematode Caenorhabditis elegans, neuronal circuits have been delineated based on patterns of synaptic connectivity derived from ultrastructural analysis.
The ultrastructural appearance of DCAMKL1 cells and the expression patterns of DCAMKL1 in normal and pathological states indicate that the cells belong to a progenitor cell population.
The ultrastructural appearance of DCAMKL1-positive cells and the expression patterns of DCAMKL1 in normal and pathological states indicate that the cells belong to a progenitor cell population.
Ultrastructural analysis using high-voltage electron microscopy (HVEM) showed bundled fibre-like patterns of GFP-Tpx2 reminiscent of microtubule structure within the apical processes.
The patterns of PrPSc deposition in prion diseases are well characterised at immunohistochemical and ultrastructural levels [ 1, 2] and most observations focus on pathological changes in the grey matter.
The choice of gene testing should be influenced more by the ethnic background of the affected child than the pattern of ultrastructural deposits.
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