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Ultrastructural analysis demonstrates progressive degradation of myelin sheaths with axonal preservation.
Ultrastructural analysis demonstrates that miR-8 is required for SSR morphogenesis.
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The ultrastructural analysis demonstrated the histological and cytological alterations occurring after pilocarpine-induced SE (Fig. 5).
Ultrastructural analysis demonstrated hypotrophy with profound myofibrillar disarray and Z-disc loss.
Moreover ultrastructural analysis demonstrated severe reduction of vesicle density in the pathologically modified mossy fiber boutons.
The ultrastructural analysis demonstrated the existence of large vacuoles within the cytoplasm of Leydig cells.
Ultrastructural analysis demonstrated mitochondrial hyperplasia and hypertrophy with frequent abnormal mitochondria and abundant mitochondrial inclusions (abundant matrical dense granules and well developed crystalline arrays; Figures 2d-f).
Ultrastructural analysis demonstrated filamentous structures organized occasionally in masses tending toward sphericity but also in bundles distributed throughout the cytoplasm (arrows in Figure 8j).
Ultrastructural analysis demonstrated the typical autophagosomes in penumbra area of WT mice after MCAO but scarce in arrb1 −/− mice (Fig. 5D).
HV antigen was only detected in the kidney and immunopositivity was clearly observed in the intranuclear inclusions bodies of tubuloepithelial cells and small amount was associated with cell debris in necrotic areas.. Ultrastructural analysis demonstrated that intranuclear inclusion bodies labeled immunohistologically corresponded to HV particles.
In all, these data corroborate the ultrastructural analysis and demonstrate severe disturbance of the triad structure with DNM2-S619L expression.
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