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Rutin (3,3',4',5,7-pentahydroxyflavone-3-rhamnoglucoside) is a flavonoid of the flavonol type abundantly present in S. involucrata.
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Although these cell types abundantly express IGF‐1 receptors and are known targets of IGF‐1 (Sonntag et al., 2013), the role of IGF‐1 in the regulation of functional hyperemia in response to neuronal activation is not well understood.
In general, areas of cell types abundantly present in selected tissue sections, such as squamous cells in non-small cell lung cancer or bronchial epithelial cells in airway tissue, allows isolation of sufficient total RNA in a relatively short time to perform only one round of amplification.
This cell type is abundantly present in intestinal tissue.
Both receptor types are abundantly expressed along the nephron and are generally involved in inhibition of transport processes (Kishore et al. 1995, Lehrmann et al. 2002, Bailey 2004, Rieg et al. 2007, Pochynyuk et al. 2008).
Cytokines derived from these cell types are abundantly expressed, and it is now commonly accepted that tumor necrosis factor (TNF) and interleukin-1 (IL-1) are pivotal mediators in the pathogenesis of RA, providing validated targets for successful therapy [ 1, 2].
Cav-1 has been found in many cell types, but is abundantly expressed in endothelial cells, type 1 pneumocytes, epithelial cells, smooth muscle cells and fibroblasts [ 2- 5].
This could be explained by the preferential affinity of H5 HA to sialic acid receptors with an α2,3 galactose (α2,3Gal) linkage dominating on the cells of the lower respiratory tract, but not to the α2,6Gal type, which is abundantly present in the human trachea [7], [8].
Although 'di-nucleotide' repeat type are observed abundantly in eukaryotes (38), on the contrast, our analysis reports 'mono' repeat patterns as the most abundant type.
C-C chemokine-receptor type 5 (CCR5) is abundantly expressed in the RA synovium and T helper-cell type 1 inflammatory infiltrates, and is bound by macrophage inflammatory protein (MIP -1α (CCL3), MIP -1α(CCL4), and RANTES (regulated upon activation, normal T cell expressed, and secreted; CCL5) [ 2].
In contrast to wild-type mice with abundantly dispersed CD8+ T cells in the challenged ear MMP19−/− mice showed low numbers of T cells that were comparable to the unsensitized situation (Fig. 2E).
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