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X-ray micro-tomography analyses reveals that two main defects are generated by the process, namely residual support material and excess of resin trapped inside the porous network.
We have confirmed that the emission wavelengths for the two main defects which make up the visible band are as follows: the oxygen vacancy emission wavelength at approximately 530 nm and the zinc vacancy emission wavelength at approximately 630 nm.
We have determined the emission wavelengths for the two main defects which make up the visible band, the oxygen vacancy emission wavelength at approximately 530 nm and the zinc vacancy emission wavelength at approximately 630 nm.
Similar(57)
The relative low elongation achieved, similar to fusion-welded sheets, is explained by the occurrence of three main defects: root opening, thinning and kissing bond.
The three main defects in diabetes, which include impaired insulin secretion, insulin resistance, and glucagon excess, will all be targeted by such a combination of medications.
This unexpected phenotype may relate to two main cellular defects.
Type 2 diabetes is a complex metabolic disease that results from two main pathophysiological defects: impaired insulin sensitivity (IS) and β-cell failure (1).
NGT 1h-high individuals may represent an intermediate state of glucose intolerance between NGT and type 2 diabetes characterized by insulin resistance and reduced β-cell function, the two main pathophysiological defects responsible for the development of type 2 diabetes.
This relationship reflects the fact that both GDM and type 2 diabetes share a similar pathophysiology, characterized by two main metabolic defects: 1) target cell resistance to the activity of insulin (insulin resistance) and 2) insufficient secretion of insulin by the pancreatic β-cells to compensate for this peripheral tissue resistance (β-cell dysfunction) (1, 3).
In this study, we provide evidence that subjects with NGT, whose 1-h postload plasma glucose is ≥155 mg/dL, have a reduction in both insulin sensitivity and β-cell dysfunction, the two main pathophysiological defects responsible for the development of type 2 diabetes.
The 14 cases that did not fall within one of the three described main defect groups were excluded from the stratified analysis due to a small sample size.
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