Exact(14)
At the same time, levels of two inflammation indicators declined in the animals' intestines.
All the above findings underscore the distinct gene expression patterns in the two inflammation models of our study.
Two inflammation models were assessed, one involving systemic LPS challenge, the other a local microinjection of fibrillar Aβ42.
Together, these findings indicate that the gene expression profiles in the two inflammation models are considerably different, likely owing to the different inflammation-inducing agents.
These criteria enabled us to identify highly relevant category groups (KEGG pathways + GO biological process categories) in the two inflammation models of our study.
However, the two inflammation samples that were positive for DEK in tissue were found to be negative for the presence of DEK protein in voided urine.
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Dynamic coincidence planar images of six tumors and two inflammations in nude mice were acquired over 4 hours immediately after injection of 25.9 MBq of [18F]FDG into the right thigh of each animal.
After exposure of the cultured epithelial cells to 10 µM poly-l-arginine for 3 hours, the intensities of two inflammation-related factors (IL-8 and RANTES) increased more than 2-fold, and two other factors (IL-6 and IL-6 soluble receptor [IL-6sR]) increased 5-fold as compared to the control (Fig. 2C).
Consistently, our data demonstrated that these two inflammation-related markers were increased in AMI patients with AKI.
In particular, we have derived a model that combines two inflammation-based parameters that results in further prognostic accuracy.
Based on microarray results, expression of two inflammation-related genes (Ifit1 and Iigp1) was measured by qRTPCR in the present study.
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