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Finally, for the first time, we investigated the treatment value of anti-MIC1 antibody against the ESCC.
Additionally, we explored the role of MIC1 in the ESCC progression and discovered the treatment value of MIC1 antibody against ESCC.
Despite such complexity, a nearly 15 years long examination of breast cancer transcriptome profiles resulted in substantial contributions into subtyping and assessment of the prognostic (providing information on the likely outcome of the cancer disease in an untreated individual) and predictive (providing information on the likely benefit from treatment) value of gene signatures.
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From the experimental data treatment, values of the maximum specific growth rate, μmax, and of the semisaturation constant, KS, were estimated (Table 3).
The compounds 4 and 5, with the urinary incontinence treatment values of 76.5% and 77.2%, respectively, showed the highest amount of deals as a scientific achievement.
Moreover, after IVIG treatment values of WBC, CRP and procalcitonin were improved.
After successful treatment, values of all patients with OSCC without bony infiltration were within the normal range.
After ZA treatment, values of all markers decreased and values of bone markers of treated and untreated groups of patients were quite similar.
Log(2)ratios were created by dividing the median of the treatment values of each probe set by the median of the control values.
The adjustment for > 10% decrease in body weight was still accompanied by significant differences in pre-treatment vs post treatment values of total fat mass % (p = 0.043) and SHBG (p = 0.028), respectively.
The secondary outcomes were changes in proteinuria, post-treatment value of glycated haemoglobin A1C (Hband) and post-treatment value of serum albumin.
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