Sentence examples for treatment mutations from inspiring English sources

Against the background of this high prevalence prior to treatment, mutations in c59 and c540 were more common in samples on day 14 after non-ACT treatment; although this was not statistically significant for either mutation (figure 1, McNemar test p = 0.375 and p = 1.00, respectively).

Out of the 105 patients who were unresponsive to diazoxide treatment, mutations in ABCC8/KCNJ11 were found in 92.

Reducing signalling through mTORC1 by pharmacological treatment, mutations, restricted calorie intake or low protein:carbohydrate macronutrient ratio enhances metabolic health and increases life span in many species up to mammals.

When measured 21 weeks after treatment, mutations in T-lymphocytes increased with doses up to 100 micrograms of 1,6-dinitropyrene, but the response was nonlinear throughout the dose range.

Not surprisingly, treatment with MMS or UV leads to a further 10-fold increase in mutagenesis, but in addition, in the region that has been rendered single-stranded prior to treatment, mutations occur in widely separated clusters of multiple mutations.

We must monitor the emergence of resistance in this enteric pathogen to differentiate between the acquisition of resistance during treatment (mutations occurring in different bacterial strains) or clonal expansion of a successful strain by person-to-person spread (identical mutations associated with a single strain).

The terms induced or repressed were used when describing the comparison on gene expression between both genotypes independently of the water treatment (mutation effect).

The genetic profiles were coded on a binary scale, with 1=presence of mutation known to confer resistance to imatinib treatment (mutation on KIT exon 9 or wt profile) and 0=absence of such mutation (KIT exon 11 mutation).

Unfortunately, crucial information about cell lines (e.g., metastasis status, hepatitis virus status, drug treatment, mutation) is often missing or ambiguous in cell line databases and relevant publications, which further confounds the selection process.

Consistent with these findings, we found that mutation of the Hdac6 deacetylase domains had the same effect as TubA treatment: mutation of deacetylase domain 1 partially disrupted Hdac6's interaction with Tip60-p400 in ESCs, while mutation of both deacetylase domains abolished this interaction.

Two misconceptions were fairly common in student answers in both treatments: mutations are an adaptive response to the environment, and traits change because of need (Table 5).