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Vertebral endplate permeability and disc metabolite transport decrease during growth and aging [ 10].
The accumulation of ROS may lead to oxidative damage to mitochondrial proteins and DNA, causing loss of electron transport, decrease in ATP production and ∆Ψm dissipation [ 39, 40].
The accumulation of ROS may lead to oxidative damage to mitochondrial proteins and mitochondrial deoxyribonucleic acid (DNA), causing loss of electron transport, decrease in adenosine triphosphate (ATP) production and ∆Ψm dissipation [ 37– 40].
In the mitochondria, ROS is generated via the electron transport chain and can accumulate as by-products, which can lead to mitochondrial proteins and mitochondrial DNA, causing loss of electron transport, decrease in ATP production and ∆Ψm dissipation [ 37– 40].
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The modal share for intermodal transport (Table 2) increases by 2.87% in tons while road-only transport decreases by 0.07%.
Formation of the DPC Cr VI) complex allows for efficient membrane transport; without the membrane, Cr VI) transport decreases ∼90%.
In contrast, transport decreased significantly with increasing CTAB concentration due to reduced surface charge and enhanced aggregation of ARS-nHAP.
The activation energy of diffusion coefficient in Case I transport decreases with increasing X, but the trend for that of velocity in Case II transport is opposite.
When the surface concentration of BV2+ increased from 2 to 40 nmol cm−2, the apparent diffusion coefficient for charge transport decreased by a factor of 14.
EIS measurements confirmed that the charge transfer resistance increases and mass transport decreases in the presence of AETD upon increasing its concentration.
For a 100 ppi electrode, the enhanced mass transport decreased from 1.26 to 1.03 at low and high rotation rates, respectively.
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