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A ring is generally provided at the intersection between the cylinder and the cone of an elevated storage silo or tank to resist a large circumferential compressive force at the intersection (the transition junction).
A T-section ringbeam, consisting of an annular plate and a vertical edge stiffener, is often provided at the intersection between the cylinder and the cone of an elevated storage silo or tank to resist a large circumferential compressive force at the intersection (the transition junction).
The cone cylinder skirt transition junction is subject to a large circumferential compressive force which is derived from the horizontal component of the meridional tension in the conical hopper, so either a ring is provided or the shell walls are locally thickened to strengthen the junction.
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These results shed considerable light on the effect of ring buckling on the load-carrying capacity of transition junctions.
Finally, the implications of the experimental and finite element results for the design of steel silo transition junctions are discussed.
However, no previous experimental study on steel silo transition junctions has been reported due to the considerable difficulties associated with testing these thin-shell junctions at model scale.
This paper is concerned with the finite element modeling of the experiments on cone cylinder skirt ring transition junctions in steel silos under simulated bulk solid loading presented in the companion paper.
These comparisons show that despite the structural complexity of steel silo transition junctions, their behavior can be satisfactorily predicted by finite element analyses taking into account a number of important factors including geometric imperfections, effects of welding and the interaction between the junction and the stored solid.
Waveguide translational symmetry is interrupted by the unavoidable presence of bends, transitions, and junctions, among others.
A series of important studies have demonstrated non-pumping functions for the Na/K-ATPase that include regulation of oncogenic transformation [39], epithelial to mesenchymal cell transition [40], tight junction formation [41] and control of the plasma membrane cholesterol distribution [42].
The assay is centered around the primary gene defect responsible for the polymorphism, a G to A transition at the junction of intron 3/exon 4 which results in a frame-shift in the resultant mRNA.
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Justyna Jupowicz-Kozak
CEO of Professional Science Editing for Scientists @ prosciediting.com