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Fish contain less Se in comparison with macroalgae, zooplankton and bivalves revealing poor or insignificant trophic transfer of Se in the marine food chain.
In contrast, there was no evidence that maternal transfer of Se was asymptotic.
In contrast, maternal transfer of Se resulted in egg concentrations similar to concentrations in the female carcass.
Maternal transfer of Se by G. carolinensis also greatly exceeded the Se concentrations maternally transferred by female reptiles, birds, and fish studied at the same site.
We hypothesized that disruption of normal development due to maternal transfer of Se and/or other elements may partially underlie these observations.
However, the ability of spider mites to tolerate 150 mg Se kg-1 may allow transfer of Se into higher trophic levels, which is an area that needs to be further investigated.
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Logistic regression suggested that females that transferred concentrations of Se ≥ 20 μg/g to their eggs had very little (i.e., concentrations BDL) Hg in their eggs.
Most notably, we described maternal transfer of high concentrations of Se that were strongly related to the concentration of Se accumulated in female tissues.
We develop a family of SE transfer circuits based on MOSFET-based SE turnstile.
This has been suggested by a recent study by Burk et al. in pregnant mice that identified two mechanisms of Se transfer; from early to mid-gestation, plasma GPx and SEPP1 were transported via the uterine fluid, probably by pinocytosis, whereas in the latter half of gestation, placental transfer of maternal SEPP1 occurred through apoE receptor 2.
This observation suggests that the reproductive strategy employed by organisms (e.g., large vs. small clutches) may have important implications for the quantity of Se transferred during each reproductive event.
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