Exact(8)
Table S4 contains the correlations between traits shown in Table 1.
Five isolates with different combinations of the traits shown in figure 2 A were chosen for detailed analysis.
All blood samples were collected in pre-chilled tubes and processed for analysis of various metabolic traits (shown in table 1) by routine clinical chemistry.
In agreement with the foliar traits shown in Table 1, the chlorophyll content was constant in P. tarapacana, regardless of elevation (Fig. 4A).
An LME examining the response of all nine traits shown in Fig. 3, to species, with site as a random effect, showed a significant difference among the three plant groups, with the hybrid significantly different from both parents (T. porrifolius: P = 0.0172, T. pratensis: P = 0.0463) (Table 1).
For the traits shown in Table 3, expressing the total s.d. of animal variation (including any due to sires and sire type) as a percentage of the mean, resulted in CVs of animal effects ranging from 4.4% (%FE on days 2 and 4) to 6.4% (DMP ignoring feed intake).
Similar(52)
In all cases, the groups did not correspond to the Al tolerance trait shown in the physiological test.
χ results for the qualitative traits and descriptive statistics for the quantitative traits are shown in Table 1 and Table 2, respectively.
The genetic and phenotypic correlations between all traits are shown in Appendix 2. Of 12 genetic correlations between body weights and survival traits, four were significantly positive, five positive but nonsignificant and three negative but nonsignificant (Table 4).
Not all phenotypic traits were measured on all individuals; sample sizes and mean values (±SE) for all traits are shown in Table 1.
When these genes were compared with genes correlated with clinical traits in the Icelandic Family Blood (IFB) study (15), DE and DC genes were enriched for different sets of clinical traits as shown in Supplementary Material, Table S5.
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