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Leaching toxicity was also determined to ensure the environmental safety of AAC with MSWI bottom ash.
The toxicity was also found to be dependent on the nanoparticles and their concentration.
Liver toxicity was also described in rats after inhalation and dermal uptake of PFCs.
Toxicity was also assessed for these substances by means of in vitro and in silico methods (the CTD discussed above is also integrated in ACToR).
No toxicity was also reported for pure silica MFI zeolite 50 and 100 nm nanoparticles [48] and LTL zeolite 80 nm nanoparticles [49].
The median lethal dose (LD50) of Te NRs and potassium tellurite (K2TeO3) were determined in mice and the subacute toxicity was also evaluated.
After stripping almost 98% ammonia of the concentration after EC experiments at 0, 5 and 120 min electrolysis times, the toxicity was also assessed.
The potentiated arsenic toxicity was also inhibited in cells stably expressing a dominant negative mutant of c-Jun N-terminal kinase 1 [JNK1].
This reduction in renal toxicity was also influenced by the time interval between the fractions; renal toxicity was significantly reduced for intervals of a week or longer compared to a 1-day interval.
In addition, solution acute toxicity was also evaluated during the treatments under optimum conditions, since some degradation by-products of 2,4-D can be more toxic than the parent compound.
Their toxicity was also dependent on the chemical nature of the polar head group.
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