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Though GO has much toxic effect, the toxicity may be avoided by modifications of GO [47, 48].
It was hypothesised that inhibition of toxicity may be stereospecific.
However, this toxicity may be reduced by increasing the nanoparticle size.
Lithium-related neurological toxicity may be severe resulting in seizures, myoclonic encephalopathy, and coma.
Bryant Day demonstrate that response and toxicity may be considered as independent (Φ = 1).
The higher toxicity may be due to the higher iron content.
The evaluation of potential toxicity may be performed by desktop screening (in silico) and (quantitative) structure-activity relationship (Q SAR).
The toxicity may be lethal to fish or can induce stress resulting in alteration at molecular, biochemical or physiological levels.
Furthermore, oxygen toxicity may be reduced as lower FiO2 often are required compared with mandatory ventilation [8].
Cd toxicity may be attributed to both acceptor and donor side of PSII thus preventing photoactivation (Sigfridsson et al. 2004).
Toxicity may be studied from the viewpoint of biochemical mechanisms understanding what molecule, pathway, or structure is "poisoned".
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