Sentence examples for toxic principles from inspiring English sources

However, some of them such as chamuvaritin, gossypol, plumbagin, and scopoletin were also documented as toxic principles of some medicinal plants [ 51].

The studies on antimutagenic activity of plants have indicated the involvement of chemical constituents which could act as nonspecific redox agents, free radical scavengers, or ligands for binding metals or toxic principles [ 36].

Toxic principles of bracken fern are known to be immunosuppressive, mutagenic, clastogenic, and carcinogenic [ 6, 7]; the most important carcinogen appears to be a sesquiterpenoid named ptaquiloside (PT) [ 8].

The toxic principle of nanosilver, that is, whether toxicity is ion or particle driven, yet has to be elucidated and analysis of the toxicological profile has to be adjusted accordingly.

The extent of the decline varies with the toxic principle involved, but it rarely makes a dangerously deep descent.

The toxic principle, urushiol, is produced in the resinous juice of the resin ducts of the leaves, flowers, fruits, and bark of stems and roots but not in the pollen grains.

Medical treatment of poisoning may include the use of activated charcoal to decrease gastrointestinal absorption of the toxic principle along with supportive care including anticonvulsant drugs such as a benzodiazepine.

So in principle toxic securities can now be carried on banks' books at whatever valuation the bank chooses while it waits for markets to revive -- a revival that may never come.

As a contribution to the search of non toxic, novel antibacterial principle from medicinal plants of Cameroon, results of in vitro antibacterial investigation is being reported herein.

Accordingly, electrophilic and nucleophilic molecules are classified as being either soft (relatively polarizable) or hard (relatively non-polarizable) and, based on this principle, toxic electrophiles will react selectively with biological targets of comparable softness or hardness.

The protein α-synuclein (αSyn), the principle toxic effector in PD, has been shown to interfere with neuronal Ca2+ fluxes, arguing for an involvement of deregulated Ca2+ homeostasis in this neuronal demise.