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In the present study, we investigate the influence of total PM and PM emitted from local traffic on the cumulative incidence of type 2 diabetes in a highly urbanized region of Germany.
In contrast, there is a stronger effect for traffic-specific PM than for total PM, when comparing estimates for the same increase in PM-mass concentration (i.e., for an increase of 1 μg/m), pointing towards a higher toxicity of traffic-specific air pollution compared to that of total PM.
Our study points to a possible effect of total PM and more specifically of PM related to local traffic on type 2 diabetes risk in the general population of a highly urbanized region in Germany.
We observed similar effects of traffic and total PM when referring to the IQR which is not surprising as this compares the PM exposures for the within population contrast in exposure.
Our study could not examine different size classes however, a detailed study of PM in Darwin during 2004 5 found that the total PM2.5 comprised on average 56% of the total PM [ 44].
Release by EE (<16%), total PM (<11%) and PM external layer (≈0) were not temperature sensitive.
The total PM contribution, from different indoor emission sources, was analysed in terms of both emission strength and duration.
The peak total PM concentrations (all sources) from 23 24 May and 25 29 May are shown in Figure 9.
Thus, opacity-based PM emission factors were based on a correction factor of 5 to approximate total PM.
The total PM and its dry soot (DS) and soluble organic fraction (SOF) constituents were analyzed corresponding to their specific fuel physiochemical properties.
BC accounted for 5 20% of total PM mass across all commute modes with an average fraction of ∼7% of PM2.5.
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