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Taken together, we propose Set2 as a key player in the DNA repair pathway that likely impinges on genome stability.
Taken together, we propose a two-step evolution model for fruit size enlargement in apple to characterize its unique evolution process (Fig. 4c).
Taken together, we propose that dysregulation of cortical 5-HT2CR may be a neurobiological mechanism underlying the intersection of impulsive action and cocaine cue reactivity.
Taking these results together, we propose that YCR041W overexpression can be used as a novel chromosome engineering tool for controlling mini-chromosome maintenance and loss.
Taken together, we propose that LNs induce autophagy-lysosome signaling and neurovascular response at least partially via an Atg5-dependent pathway.
Taken together, we propose a model in which PkaC1 phosphorylation both positively and negatively modulates its activity.
Taken together, we propose that the nucleolar localization of hTERT involves more than just the association with the hTR subunit.
Taken together, we propose that it is crucial to reduce the maintenance energy of the xylose metabolizing strains to improve xylose utilization.
Taken together, we propose skeletal muscle has a local tumor suppressive effect.
Taken together, we propose that TAK1 is essential for preventing injury-associated intestinal inflammation by the following two mechanisms.
Taken together, we propose a possible mechanism regulating Bmi-1 stability by Akt or p38 signaling (Figure 7).
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Justyna Jupowicz-Kozak
CEO of Professional Science Editing for Scientists @ prosciediting.com