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Many models have been used to study this interaction process during the past several decades.
The biobank collects these other types of information to study this interaction – how non-genomic factors affect gene expression.
Protein crystallography, used to study this interaction, has already extended our understanding of the molecular rules that govern T cell immunity.
To study this interaction and influence of microculture, we derived both spinal motor neurons and BMECs from human induced pluripotent stem cells and observed increased calcium transient function and Chip-specific gene expression in Organ-Chips compared with 96-well plates.
To study this interaction in neural tissue, co-immunoprecipitation analyses were performed in E16 brains.
To study this interaction, we used, as a Stat3-inducible model, MM1 cells.
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In future studies we plan to study this potential interaction, and we plan to investigate the interaction of the two learning systems for more complex tasks in which the dynamics of movement must be learned.
Dynamic land use and transport interaction models provide a good platform to study this mutual interaction.
We further propose a comprehensive analysis framework that combines well-characterized human cohorts and innovative in vitro model systems to study this complex interaction.
We will present the limits of these techniques to study this particular interaction and precautions that need to be taken during MAPs preparation.
Considering that δEF1 is directly associated with the E-cadherin proximal promoter in vivo, acting as a direct transcription repressor [ 36], we wished to study this physical interaction in response to BMP-6 by quantitative CHIP assay.
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