According to some studies, prenatal exposure to cocaine seems to have a negative effect on the child's neurological development and intellectual and emotional behaviour, due to the effect of cocaine on the monoaminergic system [ 34, 35].
In this review we consider methods being used to study the prenatal environment and schizophrenia, particularly the relationship between prenatal lead (Pb) exposure and schizophrenia.
Therefore, our general aim is to study whether prenatal maternal psychosocial stress is associated with an adverse cardio-metabolic risk profile in the child at age five.
Therefore, our general aim is: ▪ To study whether prenatal maternal psychosocial stress is associated with an adverse cardio-metabolic risk profile in the child at age five The corresponding research questions are: I.
From this background, we aim to study whether prenatal parental tobacco smoking is associated with DNA methylation of IGF2 differentially methylated region (DMR) and H19 in umbilical white cord blood cells of newborns, with a focus on differences in sensitivity between genders.
Our focus herein from a prospective study, prenatal exposure to PCBs may disrupt pregnancy in humans [ 4].
In that study, prenatal exposure to tobacco smoke was associated with later ADHD diagnosis, whereas postnatal exposure at home was not (Braun et al. 2006).
In a recent epidemiologic study prenatal exposure to MEP, MBP, MBzP, and monoisobutyl phthalate was associated with shortened anogenital distance (AGD) in male infants (Swan et al. 2005).
