Magnetotransport data allows us to explain this observation.
To explain this observation, we consider arsenic evaporation and consequent change in the initial substrate.
To explain this observation, samples with increasing levels of bioencapsulation (free-compound, homogenized-puree, puree) were examined.
The existence of a drop-sinking barrier, larger for a curved liquid surface, is proposed to explain this observation.
We sought to explain this observation by comparing the capabilities of various size VA and AA anastomoses to compensate for the effects of the primary AV.
To explain this observation, we consider the number of phonons (originating from vibrations of ligands) required to bridge the 5D0 7F6 energy gap (~12000 cm−1) of europium.
It is difficult to explain this observation as there was no familial history and literature does not support the occurrence of Class III in thalassemic patients.
A mechanism is proposed to explain this observation in which Na2O in the ash reacts with ambient water vapour at high temperature.
Several plausible hypotheses were proposed to explain this observation in terms of ligand flexibility and/or entropic penalties, reduced surface area available for interaction, target specific restrictions and size-dependent complexity that reduced the probability of optimal fitting.
Two possible mechanisms can be used to explain this observation.
