Sentence examples for tissue senescence from inspiring English sources

These structural signs of tissue senescence were particularly accentuated in seven of the ten subjects.

The majority of the differentially expressed NAC transcription factors (23 in leaf and 40 in cotyledon) were upregulated during tissue senescence, with 8 genes common to both organs.

While GVR treatment delayed tissue senescence, infiltration with LBR actually induced a premature yellowing and, in some cases, even necrosis starting from the point of infiltration.

This allowed us to determine whether most DNA degradation/loss takes place after tissue senescence or after deposition in the sediments.

Malondialdehyde (MDA) is a byproduct of lipid peroxidation and can be used to measure damage resulting from oxidative stress during tissue senescence [ 21, 24].

Our senescent material consists of tissues from 10 individuals from the macrofossil reference collection at the University of Wyoming, which were all collected from live trees in the field, usually at the time of tissue senescence and/or shedding of organs.

Although mycorrhizal fungi colonize plant roots and reside into the rhizosphere, endophytes live entirely within plant tissues and may develop within roots, stems and leaves, sporulate at plant or host-tissue senescence [9, 10, 11].

The current review focuses on the biological functions of Sirt1 in obesity-associated metabolic diseases, cancer, adipose tissue, aging, cellular senescence, cardiac aging and stress, prion-mediated neurodegeneration, inflammatory signaling in response to environmental stress, development and placental cell survival.

Accumulation of oxidative macromolecular damage causes cellular senescence, tissue aging and reduced life span [ 6], as well as neurodegeneration [ 24] and metabolic disorders [ 25], which are diseases associated with aging.

In recent years, the role of telomere length in the pathology of cardiovascular disease (CVD) and diabetes, where tissue ageing and senescence play major roles, has attracted a continuously growing research interest, and in the last two years alone six articles on telomere length have been published in Atherosclerosis.

The association between cellular senescence and tissue ageing seems to be causative, since lack of p16, which precludes senescence establishment, prevents the age-related decline, thereby increasing healthspan [ 25 27].