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GRC samples thus treated, with various moisture contents, were mechanically tested before and after a period of accelerated ageing.
Adult male C57BL/6 mice were thus treated with clinically relevant doses of lopinavir/ritonavir for 4 weeks, and subjected to thorough metabolic, neurobehavioral, and biochemical analyses.
The paraaortal lymph nodes were thus treated with higher degree of suspicion than other distant sites, with regard to likelihood of metastasis.
They were thus treated with topiramate which induced a significant improvement in one patient and a partial response in the other two.
Enterocytes were thus treated with human PrPc-siRNA, before the onset of cell polarity and the formation of cell cell junctions.
BT-474 and SK-BR-3 cells were thus treated with DZNep/TSA in the presence or absence of Aza for 72 h and the changes in CDKN1C expression were determined by quantitative RT-PCR analysis.
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We thus treated cells with MG132 and performed coimmunoprecipitation with overexpressed Flag-HIF-1α in normoxia (Fig. 3C, left panel), since the pVHL- binding sites of all HIF-αs are conserved.
We thus treated DC with IFNα and LVP to simulate an in vitro therapy.
We thus treated HMEC1 with media conditioned by control or vGPCR-expressing cells and assessed the activation of TSC kinases by these supernatants.
To further substantiate the existence of such a precise window we thus treated embryos with 2.5 µM of genistein at 5 hpf or 20 hpf and monitored apoptosis 7 hours after exposure (Figure 2A D).
We thus treated HMEC1s with supernatants derived from vGPCR-expressing cells (vGPCR CM) or control cells (Control CM), in the absence or the presence of the mTOR inhibitor, Rapamycin.
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