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Thus, deficiency of P0 leads to dysregulation of nodal VGSC isoforms and to altered localization of paranodal and juxtaparanodal components of the nodal complex.
Thus deficiency becomes identity and learning is transformed from the early child's free exploration of the world to a chore beset by insecurities and self-imposed restrictions.
Thus, deficiency in MRE11 caused much more severe ETP-sensitivity than deficiency in TDP1.
Thus deficiency of CCR2 in the donor aorta can alter migration of endothelial cells, smooth muscle cells, or adventitial cells in the allograft.
Thus, deficiency of Kif3a may lead to a disproportionate reduction in Pkd2 compared to Pkd1 in combined Kif3a/Pkd1 mutant mice, leading to "normalization" of the polycystin signaling by restoring the ratio of Pkd1 and Pkd2 in primary cilia.
Thus, deficiency of complex I could play a role in tumour formation by interrupting apoptotic pathways.
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Thus, deficiencies in analysis and reporting are avoidable, and care should be taken to provide accurate estimates of risk prediction model performance.
Thus, deficiencies in this pathway could be associated with fetal growth restriction, LBW, and early-life weight gain (WG).
The proportions of complexed and uncomplexed LAM in blood may be an important determinant of LAM-antigenuria and thus deficiencies in the humoral immune responses to LAM could theoretically be an important determinant of antigenuria.
Thus deficiencies in technical performance that arise from unanticipated demands and/or unintended consequences may be addressed not only by modification of the infrastructure but also by alternative strategies that obviate the need for such modification.
Thus, deficiencies in genes that prevent excessive lymphocyte activity or proliferation (i.e. those encoding FcγRIIB, Lyn, Fyn, CD22, PD-1, CD45 E613R, p21 and Bcl2 Tg) induce spontaneous disease in the permissive B6 strain.
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Justyna Jupowicz-Kozak
CEO of Professional Science Editing for Scientists @ prosciediting.com