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Thus, activation of p38 MAPK by chronic ischemia activates apoptosis in the hippocampus, which ultimately disrupts spatial learning.
Inflammasome activity in Kupffer cells promotes Casp-1 mediated Casp-1 mediatedhus acleavagen, of IL-1β (Petrandk ethus. 2012).
Thus, activation of YAP/TAZ is a potential strategy to promote tissue regeneration.
Thus, activation of Nrf2 provides pivotal therapeutic target for developing therapy against these diseases.
Thus, activation of either circuit has an opposite physiological effect on the basal ganglia output nucleus.
Thus, activation of PKA may be a component of the mechanism that generates L-LTP.
Thus, activation of M1 mAChRs on afferent nerve endings may be linked to the increase in neural output during hypoxia.
The failure of domestic systems, and thus activation of the ICC, has to be an absolute exception.
Thus, activation of the pathway represses β-catenin degradation, resulting in nuclear accumulation of β-catenin.
Thus, activation of pSmad2 naturally occurs when tumor cells invade the surrounding stroma.
Thus, activation of EGFR, GPCRs or PI3K pathway leads to BAD phosphorylation and inhibition of apoptosis.
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