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The ectopic Purkinje cells in the NZ take two forms–scattered randomly throughout the granular layer and intralobular white matter, and in reproducible clusters misaligned from the Purkinje cell layer (arrowheads-Fig. 5N).
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Our results show that ABCA12 localizes throughout the entire Golgi apparatus to LGs at the cell periphery, mainly in the granular layer keratinocytes.
The spinous layer is succeeded by the granular layer, or stratum granulosum, with granules of keratohyalin contained in the cells.
The granular layer is idealized as Pasternak shear layer [26].
Furthermore, the granular layer as a whole gradually tilts.
In contrast, the choline uptake rate in the granular layer eventually decreased slightly.
The two yellow lines trace the surface of the granular layer and the two-layer interface.
The granular layer beneath the strip footing idealized as incompressible shear layer.
Within the granular layer, a mossy fiber generates a series of enlargements called rosettes.
Pofut1 conditional inactivation led to ultrastructural abnormalities in the granular layer and altered filaggrin processing in the epidermis, suggesting a perturbation of the granular layer differentiation.
On the other hand, Golgi cell dendrites begin within the granular layer and extend into the molecular layer and can therefore receive presynaptic input from GCs both within the granular layer and in the molecular layer.
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