Early detection of infection is very crucial for efficient management of early blight of tomato caused by Alternaria solani.
Fusarium wilt disease of tomato caused by Fusarium oxysporum f. sp. lycopersici is one of the most devastating disease worldwide which causes yield reduction and affects tomato production (Hanaa et al. 2011).
FA infiltration in the leaves of tomato caused damage to DNA by producing cleavages which was visible in the form of degraded DNA on agarose gel, whereas, the DNA degradation was completely absent in the control leaf tissues (Figure 8). Figure 8 DNA quality of control as well as FA-treated tomato leaf tissues.
The ectopic expression of the Ginkgo AGAMOUS gene in tomato caused the homeotic transformation of the transgenic sepals into carpel-like structures, and this showed that the gymnosperm gene has a genuine C function.
The inhibition of its perception or biosynthesis in tobacco and tomato caused delayed onset of leaf senescence and lower expression of SAGs, which was also found in Arabidopsis ethylene-insensitive mutants [ 158– 158].
In fact, ectopic expression of AGAMOUS in tomato caused sepals on greenhouse-grown plants to swell and lose chlorophyll, and, upon placing these plants at 16 18°C°C, the sepals accumulated lycopene [ 28, 23].
Reduced branching due to TB1 overexpression was critical to maize domestication; conversely, the knockout of BRC1 expression in Arabidopsis and tomato causes increased branching.
Similarly, AAL toxin, a host-selective toxin produced by Alternaria alternata f. sp. lycopersici (a pathogen of tomato) causes cell death in both plants and animals and appears to target the same step in ceramide biosynthesis as fumonisin B1 [ 8, 9].
The yield increase in tomatoes caused by heterozygosity at SFT is due to suppression of growth termination imposed by the SELF PRUNING (SP) gene (Krieger et al. 2010).
Fuhrman et al 37 showed that 60 mg of lycopene per day for 3 months in six men (approximately equivalent to the amount of lycopene in 1 kg of tomatoes) caused a 14% reduction in plasma LDL-C with no significant change in HDL-C.
