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This response involves intentionally making things slightly different to completely different, forcing an attacker to alter a standard attack vector, tactics, and methodology.
In C. elegans this response involves the chaperones mtHSP70/HSP-6 and HSP-60.
This response involves the cycling of SCs between distinct differentiation states that support nerve regeneration.
This response involves mechanosensing, which in macrophages depends on Rac1-mediated signal transduction [ 4].
This response involves an mRNA levels increase of amino acid biosynthesis genes and especially oxidative stress related genes.
This response involves multiple regulatory networks which translate signals to alter the reorganization of the bacterial cell to survive unfavourable conditions [ 3].
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This response involved both taxonomic and functional diversity.
This response involved powers of intervention unknown during normal times, and the actions generated widespread fear and resentment.
Inflammation is part of this response, involving signalling via cytokines and via activation of the complement system to recruit the immune cells to the site of stress.
Signaling for this response involved the phosphorylation of the serine-threonine kinase Akt by the DNA damage signaling kinase DNA-PKcs, and NF-κB activation [ 6].
This inflammatory response involves the recruitment of macrophages and neutrophils to the damaged pancreas and to the surrounding adipose tissue.
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