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The investigators concluded that pressure overload triggers cardiac autophagy, and this response contributes to progression of heart failure.
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Unexpectedly, this increased inflammatory response contributes to the progression of atherosclerosis but not insulin resistance.
We propose that this sustained inflammatory response contributes to the neurological deficits we observe in adults following larval irradiation (Sudmeier et al. 2015).
This aberrant immune response contributes to the physiopathology of diseases such as food allergies, celiac disease (gluten immune response), and inflammatory bowel diseases (IBD) including ulcerative colitis (UC) and Crohn's disease (CD).
This excessive proinflammatory response contributes to the pathogenesis of severe influenza pneumonia and may partly explain the high mortality rates in young, otherwise healthy adults during the pandemic of 1918 to 1919 [ 13, 14].
Therefore, both Th1 and Th2 response contribute to the initiation and maintenance of AD.
This observation suggested that nucleosomal response contributes to ERK/MAPK mediated transient induction IE genes critical for neuronal plasticity in those brain regions.
This lack of serial mediation effects further corroborates that each brain response contributes independently to the translation of noxious stimuli into the different pain dimensions.
Laguesse, S. et al. A dynamic unfolded protein response contributes to the control of cortical neurogenesis.
The CD40-mediated immune response contributes to a wide variety of chronic inflammatory diseases.
Recent evidence demonstrates that neuroinflammation and immune response contributes to the development of neuropathic pain.
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