This process is regulated by a TemplateReactionRegulation that contains mostly a transcription factor as regulator.
This process is regulated by miR-22 [130,131].
It was believed that this process is regulated by protein kinase-mediated phosphorylation/dephosphorylation of CAR.
As mineralized cartilage does not contain any cells, it is unclear how this process is regulated.
This view of ever-changing NPCs necessitates the re-evaluation of current models of nuclear transport and how this process is regulated.
Availability of substrate for this process is regulated, in part, by N-Acyl-D-glucosamine 2-epimerase (NAGE), which interconverts N-acetyl-glucosamine (GlcNAc) and N-acetylmannosamine (ManNAc).
This process is regulated by the external pore openings in a particle; the second, elongated step on the deswelling curve represents the evaporation of the solvent from the polymer network.
This process is regulated in multiple ways at different stages of gene expression.
It is largely unknown how this process is regulated in the human pathogen Mycobacterium tuberculosis.
This process is regulated by the balance between positive and negative inputs in the local environment.
Characterization of these intracellular Zn pools is critical to understanding how this process is regulated.
