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This process is orchestrated by signaling molecules that govern cell proliferation, migration, and differentiation, which depend on the microenvironment and recruitment of progenitor cells, recapitulating embryonic development [[4]].
It follows that cells undergoing mesenchyme-to-epithelium transitions must reorganize their microtubule network extensively, yet little is understood about how this process is orchestrated.
The complex interplay of heterogeneous reasoning modes that is involved in this process is orchestrated by a special first-order logic system that uses static abstraction levels, dynamic declarative meta control, and a simple form of truth maintenance in order to test models quickly and cheaply.
This process is orchestrated at the molecular level by the expression of the myogenic basic helix-loop-helix (bHLH) transcription factors MyoD, Myf5, myogenin, and MRF4[1].
This process is orchestrated by a specialised cytoskeletal array termed phragmoplast that delivers those membrane vesicles to the division plane.
This process is orchestrated by a splicing cascade relying on phosphorylation/activation of the SAM68 by the extracellular regulated protein kinases (ERK1/2).
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This process was orchestrated by IFNAR1 expressed on both immune and tissue-resident radioresistant cells.
This fundamental process is orchestrated via specialised chromatin structures termed centromeres.
This growth process is orchestrated by various actions at the growth plate, a developmental region consisting of chondrocytes in distinct cellular zones.
The different steps of the process are orchestrated by >30 ATG genes.
The trimming process is orchestrated by a trimming plan which consists of sequenced trimming tasks.
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Justyna Jupowicz-Kozak
CEO of Professional Science Editing for Scientists @ prosciediting.com