This clinical presentation correlates with the histologic distribution of amyloid deposits.
This variability of imaging presentation correlates with the heterogeneous histological appearance of NSIP.
Indeed, with isolated exceptions [6], disease presentation correlates well with a depression in the host's cell- mediated immunity late in gestation [7], [8].
The presence of cells with low-VEGFR presentation correlates well with immunohistochemistry studies that have reported nonuniform VEGFR expression patterns on tumor vasculature.
Advanced renal failure at presentation correlates with an increased risk of end-stage renal failure and death, and the aims of therapy are both to control disease and recover organ function [ 6].
The number and location of lesions on initial presentation correlated with the received subcutaneous injections.
CA 15-3 levels at presentation correlate with stage of disease, tumour size, lymph node status, the presence of metastases and lymphocyte infiltration into the tumour.
In both groups, the PE from each stimulus presentation correlated significantly with the BOLD midbrain signal, and there were no differences between groups in the striatal PE signal.
Wound fluid pro MMP-9 and pro MMP-9–to–TIMP-1 rando at pro MMP-9 to TIMP-1ated significantly with WHR4 weeks (r = 0.4538, pro MMP-9 to TIMP-10.4959, P < 0.0001, ratioctively).
In addition, biomarker levels at the time of presentation correlated with SOFA score at 24 hours: sE-selectin (0.37), sFlt-1 (0.64), sVCAM-1 (0.22), and PAI-1 (0.51), P < 0.001 for all comparisons; except sICAM-1 (0.13), P = 0.08.
