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This phenotype is observed in BicD and egl mutants.
Given that this phenotype is observed in the regions in which the JAK/STAT pathway is normally active, this suggests that pathway signalling is involved in the establishment of wing disc folds.
In contrast, this phenotype is observed in ∼50% of monkey Apale spermatogonia and thus comprises only 25% of the total undifferentiated spermatogonia (Adark and Apale) in the adult testis (Fig. 7, top).
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Interestingly, this phenotype was observed upon both ubiquitous and glial-specific snr1 knockdown.
This phenotype was observed regardless of which ade6 strain carried the atf1Δ allele (data not shown).
This phenotype was observed in 76% of k-ras-MO1 injected embryos (75.8%±9.8, n>500, from 15 independent experiments).
This phenotype was observed in all five HSP90-RNAi lines (48% total frequency; p<<0.001 versus control, Fisher's Exact Test) but not in controls or in HSP90-TDNA lines.
This phenotype was observed in both heterozygous and homozygous individuals.
This phenotype was observed after 50% knockdown of chd2.
Interestingly, this phenotype was observed in our cell imaging experiments.
This phenotype was observed at the earliest in 25-day-old mice.
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