This model proposes that sex serves many functions, including affiliation, play and dominance, and that reproduction is not the only or even the main purpose of sexual behavior.
This model proposes that NucS has an archaeal origin and emerged as a combination of two independent protein domains with complex evolutionary history.
For instance, this model proposes that haemophilia involves a failure of platelet-surface FXa generation, leading to a lack of platelet-surface thrombin production.
This model proposes that reduction of fitness in hybrids generally occurs due to interaction between at least two epistatic loci derived from divergent parents.
In particular, this model proposes that activity in the ventral parietal cortex (VPC), which includes the temporo-parietal junction (TPJ), reflects the capture of bottom-up attention by incoming information, either from the senses (perception) or from long-term memory (retrieval).
This model proposes that modular proteins are composed of individual folding domains that contribute distinct functionalities.
This model proposes that experiences of illness and symptoms are influenced by biological, psychological and social factors.
This model proposes that selection favours optimal codons over minor codons, while mutational pressure and genetic drift allow the minor codons to persist.
This model proposes that individuals construct schematic perceptions of illness and health-threatening conditions according to sources of information that are available to them.
This model proposes that genetic coding arose de novo from complementary base pair interactions between tRNAs and single-stranded RNAs present in the immediate environment.
This model proposes that overproduction of Aβ, or an increase in Aβ 1-42) relative to Aβ 1-40), is causal in the disease process.
