This was initially attributed solely to the higher carrier fluid refractive indices associated with higher protein concentrations.
This was initially attributed to neurotoxic effects of anaesthetics/sedatives but there is an emerging consensus that the primary insult is the inflammatory trauma of surgery in the older patient inducing acute or lasting cognitive deficits.
The behavior of this system was initially attributed to stress-induce mutagenesis (Yang et al. 2001, 2006).
At the 10 mg dose level (5 days every 2 weeks), a patient was observed to have a dose-limiting grade 3 bilirubin elevation, and this event was initially attributed to drug.
While this behavior was initially attributed to stress-induced mutagenesis (Yang and Polisky 1993; Yang et al. 2001), it can be explained by selection alone acting on mutants that arise during pregrowth and initiate clones that grow and adapt under selective conditions.
The efficacy of this procedure has been initially attributed to the bone block, which increases the anteroposterior diameter of the glenoid.
This reduced risk was initially attributed to inadequate adjustment for risk factors such as tobacco smoking [ 4, 5].
The zonation of foundation species along this physico-chemical gradient was initially attributed to physiological responses to stress and nutrient requirements [8], [23], [31].
In this patient, worsening hypertension was initially attributed to the use of immunotherapy, specifically tacrolimus and corticosteroids.
These erroneous explanations for symptoms are reflected in this patient, whose symptoms were initially attributed to abruption and who was not diagnosed with tuberculosis until >3 weeks after seeking medical assistance.
The initial response was initially attributed to a direct algogenic effect of formalin on the nociceptors whereas phase two was associated with the release of local endogenous mediators responsible for sensitization of primary and spinal sensory neurons and subsequent activation of the nociceptors [ 24].
