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The AcuI− mutant was significantly more sensitive to acrylate than the wild type parent and we showed that this hypersensitivity was overcome by reintroducing pBIO1918, a plasmid that expresses functional acuI (Figure 6).
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This limitation was overcome using haploid genotypes.
This hypersensitivity was significantly reduced by previous treatment of animals with gabapentin and also with MECM.
This hypersensitivity was associated with central and peripheral neural sensitization [ 19].
These pmr1 pmc1 cnb1 triple mutants are hypersensitive to high Na+ levels but this salt hypersensitivity can be overcome by heterologous expression of an endoplasmic reticulum-located Ca2+-ATPase (Anil et al., 2008).
Lisi was overcome.
This hypersensitivity is associated with an abundance of chromosomal damage and induction of apoptosis and necrotic cell death (3).
This is overcome by this paper.
But this is being overcome.
However, some think this can be overcome.
When this resistance is overcome, illness ensues.
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