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NvSoxB(2) is also expressed in these cells, but this expression appears to be independent of NvAth-like and might occur at a later point in the developmental program of the progenitor population.
This expression appears to be regulated by the interaction between CCAAT/enhancer-binding protein α (C/EBPα) and the promoter region of the Ces1d gene to enhance its transcription (Wei et al., 2005).
This expression appears prior to segmentation (unpublished data).
This expression appears to be a misuse of language.
At a protein level, this expression appears to be more nuclear in EAC than in BE.
CCM treatment caused reexpression of maspin, and this expression appears to be further enhanced by the combined DHA + CCM diet.
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This is good, as the expression appears to be ramping up.
This boundary expression appears to be important to prevent homeotic displacement of node and lateral organ fates into underlying stem tissue.
Also, this VEGF expression appears to be also associated with COX-2 and HIF-1 expression and activation in TAT.
This sustained antigen expression appears important for supporting the activated phenotype of transgene-specific T cells following rAd immunization [65], [66], although maintenance of memory T cell populations eventually becomes antigen-independent [66].
This sIL-18Rβ expression appears regulated during CIA.
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CEO of Professional Science Editing for Scientists @ prosciediting.com