This cardiac muscle beats throughout life with its own automatic rhythm.
To test this, cardiac injury was induced by coronary artery ligation in adult C57BL/6 mice (Fig. 1e).
Daily Coumadin, which he has been taking for about eight years, greatly reduces his chances of suffering a stroke as a result of this cardiac problem.
In the present review, we summarize the current and limited understanding of the cellular mechanisms underlying this cardiac anoxia tolerance.
Third, those samples whose two gradients satisfied a pre-set value are taken as candidates for the R peak, and the sample with the minimal included angle is regarded as the R peak in this cardiac cycle.
Our study therefore uncovered a novel molecular mechanism for cardiac hypertrophy and indicated miR-328 as a potential therapeutic target for this cardiac condition.
Since mitochondrial dysfunction may affect the epigenetic landscape, we hypothesize that this cardiac toxicity may result from epigenetic changes related to disruption of mitochondrial function.
This study was designed to confirm this cardiac protection by use of a dog CPB model and to elucidate the related mechanism.
Despite the importance of ST-segment elevation myocardial infarction (STEMI) in China, no nationally representative studies have characterised the clinical profiles, management, and outcomes of this cardiac event during the past decade.
We investigated the prognostic value of this cardiac tissue in short-term patient mortality.
This cardiac activity can be reviewed and interpreted in real time using mobile smartphone networks and cloud storage.
