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This analysis uncovers some new relationships between disorders and predicted complexes that may take a potential role in the prediction of multi target drugs.
This analysis uncovers a connection between methylation and early embryonic transcription at a major demethylation target, further emphasizing the importance of genome-wide approaches to explore methylation reprogramming and its functional impact.
This analysis uncovers a counterintuitive case in which female-biased or male-biased contributions in the same direction occur both from Africans and from Europeans in a manner consistent with estimated mean X-chromosomal and autosomal admixture fractions in African-Americans, rather than an African female bias and a European male bias.
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This analysis uncovered 94 genes (141 sequences) with differential pre-treatment expression.
This analysis uncovered only two point mutations that were observed on plasmids from Jurkat-AID cells.
Thus, this analysis uncovered unanticipated limitations regarding rad61 bypass of Ctf7 function but confirm rad61-bypass of ctf7 null cell lethality at a wide range of temperatures.
This analysis uncovered person-specific diversity in histone variants and in their PTMs, and also demonstrated a requirement for additional histone separation that should complement sequencing of peptide digests to achieve comprehensive mapping of all histone isoforms.
This analysis uncovered numerous epicardial signature genes such as uroplakin 1b, 3b, glycoprotein m6a, dermokine or basonuclin that have not previously been implicated in the function, homeostasis or regeneration of the epicardium or the heart.
This analysis uncovered 897 genes (of the 2331 differentially expressed genes in the discovery set, Figure 1) that had single nucleotide polymorphisms (12,276 total SNPs) that were statistically different between YRI versus CEPH populations (p value<1.25E-07, Bonferroni's corrected p value of 0.01, Table S2).
This analysis uncovered a strong reciprocal relationship between transcriptional strength and axis protein enrichment.
This analysis uncovered 26 double mutants with similar drug sensitivity to wild-type cells (Fig. 5B).
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Justyna Jupowicz-Kozak
CEO of Professional Science Editing for Scientists @ prosciediting.com