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Moreover, and maybe more importantly, this analyses shows that the majority of these immune responses are indeed predictable using advanced bioinformatics methods for pan-specific HLA-peptide binding and that cellular responses are hence directly explained in terms of peptide binding to one of the patients HLA molecules in accordance with earlier work by for instance Hoof et al. [28].

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This analyses show the expected order of coefficients: rxy = .05 (with one review), rxy = .11 (with two reviews) and rxy = .17 (with three reviews).

This analyses show that once the epigenetic changes to H3K9Me3 are established they remain stable and unaffected by the BXB-ER activation status.

Results of this analyses show that the proliferation signature has a high AUC compared to the clinical parameters in all data sets (Supplementary Information Table S3).

In this study, the analyses show that virtually all the oil samples used have been degraded.

The results of the analyses show this approach.

The results from this sensitivity analyses showed that the combined coefficients of endotoxin exposure and for early life dog ownership are statistically significant when excluding LISA from the meta-analyses but not significant when excluding PIAMA or INMA.

Confirming this, immunoblot analyses showed that the knock down of TRIM25 inhibits the degradation of the larger MAVS isoform as well as the ensuing phosphorylation of IRF3, but not of IκBα after activation.

Despite this, both analyses showed changes in HD relevant regions (in line with a priori hypotheses) with most prominent DTI changes located in the ventral striatum and globus pallidus.

During this period, chimerism analyses showed that the fraction of host cells was between 1 and 10%.

In this study, immunodepletion analyses showed that transcription from XCPE1- or XCPE2-containing promoters use at least RNA Pol II, TFIIB, mediator, and TFIID present in the NE as critical factors.

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