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Whether this abnormality reflects abnormal functional activity in the subcortical structures subserving tonic pain activation remains conjectural.
This suggests that the organizing signaling cascades in the NAcs of patients with schizophrenia may be abnormal and that antipsychotics might reverse this abnormality.
Can you help us find others who share this abnormality?
This abnormality was specific to the identity related targets.
This abnormality was not seen in control embryos.
We knew that this abnormality created an enzyme that caused the uncontrolled growth of cancer cells.
Drugs that boost the chemical messenger norepinephrine could alleviate some of the cognitive problems that stem from this abnormality.
This study attempts to establish whether this abnormality is also present in unaffected relatives of patients.
Correction of this abnormality by gene transfer might improve cardiac function.
This abnormality started ≤2 minutes before the onset of clinical symptoms and persisted for minutes after clinical resolution.
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The prevalence of this abnormality varies among ethnic groups [ 3].
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