KL-deficient mice show extensive and accelerated arteriosclerosis in association with medial calcification of the aorta and both medial calcification and intimal thickening of medium-sized muscular arteries [ 26].
Atherosclerosis, the most common form of arteriosclerosis (generally called hardening of the arteries), is the thickening of large and medium-size arterial walls by cholesterol deposits that form plaques, causing the size of the arterial lumen to diminish.
There was a medium effect size difference seen between BP1 and BSD subjects in the right supermarginal gyrus and right precuneus which was reflected in comparison with control subjects: BSD has a medium effect in the thickening of the right supermarginal gyrus, whereas BP1 subjects had a medium effect of thinning to the right precuneus.
Secondary thickening of the wall of cells cultured in the induction medium was confirmed by microscopic examination and correlated with an increase in the lignin content and changes in the polysaccharide composition of the cell wall.
An abnormal thickening of the tendon sheath with enhancement after intravenous administration of gadolinium contrast medium may be seen.
In humans, atherosclerotic lesions occur in the large and medium sized arteries and are characterized by an asymmetrical focal thickening of the innermost layer of the arterial wall, the intima.
Dysfunction of the endothelium and the presence of macrophages and activated lymphocytes within the vessel wall lead to thickening of the intima and media of the vessel wall of large- and medium-sized muscular arteries [ 29].
Specifically, elongated axes and paraclades lack thickening, thickening of short aerial stems results from the primary thickening meristem and/or the secondary thickening meristem.
Thickening of rhizomes results from the activity of the primary thickening meristem.
AA was defined as localized intimal thickening of ≥3 mm.
