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The GALT genotypes identified in these samples illustrate two important points.
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Detailed investigation of the failure mechanism in these samples illustrated that trabecular bone often fails in delamination, providing a mechanism for energy dissipation while conserving trabecular bone architecture.
Nevertheless, even these small samples illustrate ambiguity within records and inconsistencies across reports of the same trial.
Epifluorescence images of the bacterial/pyrite samples illustrate these differences in bacterial attachment (Figure 2).
The magnified seed and feed interfaces of both samples illustrate the variations of the interface shape at the initial and final stages of growth under μG and 1G.
The magnified images of indium mapping at the seed and feed interfaces of both samples illustrate the variations of the interface shape at the initial and final stages of growth under μG and 1G.
Likewise, the duplicate samples illustrate the reproducibility of the dust preparation and analytical methods within each sample batch.
Regarding HNSCC (head and neck squamous cell carcinoma), studies in mouse models and human samples illustrate that disruptions in TGF-β signalling promote epithelial carcinogenesis [ 4, 6– 8].
Detection of these changes in blood samples illustrates the important role of TIMP-1.
The broader intensity plots of the EC24T0 samples illustrated that there was a greater range of transcriptional change in these samples compared to the reference than observed in the SAT0 samples.
Recent whole-genome sequencing of cohorts of hundreds of patient samples illustrates the challenges of identifying driver mutations outside the exome46,47.
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Justyna Jupowicz-Kozak
CEO of Professional Science Editing for Scientists @ prosciediting.com