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Dispute transformation and resolution in these models display two interdependent but closely interconnected dimensions.

This increase in performance is likely to arise from the fact that these models display uncorrelated resampling profiles, i.e. the predictions calculated on the hold-out folds during cross-validation are not correlated (Figure 6). Figure 6 Pairwise Pearson correlation for the cross-validation predictions across the model library.

These models display hallmarks of ALS that include an age-related decrease in swim endurance, atrophy of locomotor muscles, paralysis with concomitant loss of NMJ integrity and, in some cases, early death.

Mouse aneuploidies have been engineered to model the various DS phenotypes; these models display some behavioral, anatomical and cellular abnormalities similar to human phenotypes (reviewed in Das & Reeves, 2011).

Although these models display overall similarities in the assayed characteristics, the absence of caspase homologs and classical mediators of apoptosis, such as Bid, Bax, AIF, Apaf-1 and Bcl-2 proteins, alludes to different mediators being involved in the process of CQ-induced PCD in P. falciparum.

With regard to the considerations discussed above, transgenic overexpression of Akt leads to a decrease in mitochondrial markers in skeletal muscle, both in aging mice and young mice on high caloric diet (Izumiya et al., 2008), yet both of these models display marked metabolic improvements following transgene activation.

Some of these models displayed chitin synthase inhibition in the millimolar range.

These models displayed cognitive impairment associated to hyperphosphorylated tau protein and formation of tangles [ 84- 86].

SNP calling procedures were assessed with reference to Support Vector Regression with linear kernel (SVR-lin) and Ridge Regression BLUP, since these models displayed higher predictive accuracies than other genomic selection models in following analyses.

However, these slip models display a large variability, which may reflect the non-uniqueness of an under-determined inverse problem of solving the slip distribution over a finite fault (e.g., Simons et al., 2011).

Importantly, the literature review indicates these cellular models display genetic alterations representative of those found in clinical tumors of the correspondent histotype.