Sentence examples for these effects support from inspiring English sources

Taken together, these effects support our observation that even on a NPC level, spectra have broadened significantly through time in a cellular environment.

Collectively, these effects support the preventive medical management strategies that are recommended in the tables.

Both of these effects support the other and thus achieve the best results; this holistic approach is a prominent feature of TCM.

These effects support the premise that sensorimotor contingencies can be learned with the aid of a sensory augmentation device even in later stages of the lifespan and that mastery of new sensorimotor contingencies leads to perceptual changes.

We propose a new pathogenetic mechanism for these effects supported by a MEDLINE search using the search terms metabolic syndrome, insulin resistance, diabetes, dyslipidaemia, hyperglycemia, weight gain, sympathetic, histamine, serotonin, dopamine, triglycerides, cholesterol, hypertension, cardiovascular disorders and sudden cardiac death, cross-referenced with the term schizophrenia.

These small effects support the general validity of this low-stakes assessment in Germany.

These behavioural effects support those previously observed in Australia and suggest that the intervention is a viable health promotion tool in the UK.

These compound effects support our previously proposed hypothesis [30] that local patterns of biophysical stimuli create a type of positional information that contributes to the correct patterning of emerging tissues in the joint.

These two beneficial effects support the World Health Organization concept that lifestyle interventions in obesity and type 2 diabetes have the potential to counteract the consequent social burden of depression (31).

Collectively, these Sema4A-mediated effects support the presence of a positive loop between components of innate immunity, such as stimuli, cytokines, monocytes/macrophages, and cells from the synovial stroma, such as RASFs, which may be critical to the exacerbation of inflammation in RA.

The specificity of these mutational effects supports the model that Tat interacts directly with AFF4.